2 edition of Effect of glyceryl trinitrate on pulmonary vasculature of anesthetized dogs found in the catalog.
Effect of glyceryl trinitrate on pulmonary vasculature of anesthetized dogs
Alan L. Pinkerson
by Federal Aviation Agency, Office of Aviation Medicine in [S.l]
Written in English
|Statement||by Alan L. Pinkerson, Peter A. Kot, and Donald M. Knowlan.|
|Series||AM -- 64-11., AM (United States. Office of Aviation Medicine) -- 64-11.|
|Contributions||Kot, Peter A., Knowlan, Donald M., United States. Office of Aviation Medicine., Civil Aeromedical Research Institute (U.S.)|
|The Physical Object|
|Pagination||p. [18-20] :|
|Number of Pages||20|
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Effect of glyceryl trinitrate on pulmonary vasculature of anesthetized dogs 1 v. (OCoLC) Material Type: Document, Government publication, National government publication, Internet resource: Document Type: Book, Computer File, Internet Resource: All Authors / Contributors: Alan L Pinkerson; Peter A Kot; Donald M Knowlan; United States.
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Effect of glyceryl trinitrate on pulmonary vasculature of anesthetized dogs by: Pinkerson, Alan L. Published: () Extraction of nitroguanidine (NQ) from triple-base gun propellant by: Morris, Jeffrey B.
Published: (). The effect of nitroglycerin on pulmonary vascular capacitance in dogs In this study we investigated the hypothesis that the decrease in pulmonary vascular pressures observed after administration of nitroglycerin is in part due to a shift in the pulmonary vascular pressure-volume by: 4.
Effect of Nitroglycerin On Pulmonary Perfusion Distribution and Gas-exchange of Normal Subjects WA Briscoe, and E. Carlsen, The lung: clinical physiology and pulmonary function tests, 2nd ed.
Chicago: Year Book Medical Publishers, Knowlan D. M., Effect of Glyceryl Trinitrate on Pulmonary Vasculature of Anesthetized Dogs., 10 Cited by: 3. The effects of nitrites and xanthines on coronary inflow and blood pressure in anesthetized dogs.
Amer. Heart J. 21, – (). Google Scholar Brachfeld, N., Bozer, J., Gorlin, R.: Action of nitroglycerin on the coronary circulation in normal and in mild cardiac by: The most prominent pharmacologic effects of the nitrate-nitrite drugs are manifested on the vasculature; the small blood vessels, arterioles, capillaries, and venules.
The details of these actions. Pinkerson AL, Kot PA, Knowlan DM: Effect of glyceryl trinitrate on pulmonary vasculature of anesthetized dogs. Scarborough WR: Comments on progress in ballistocardiographic research and the current state of the art.
AD Gogel WC: The. Moreover, HNO has been shown to elicit a vasodepressor effect in vivo, lowering blood pressure in anesthetized rabbits (18) and cats (6), and conscious dogs (23,24) and rats (12). In dogs, at. This chapter focuses on the mechanism of action, dosing, and pharmacology of midodrine, adrenergic agonists and antagonists.
Midodrine is a selective alpha-1 agonist and results in alpha. Studies on the glomerular microcirculatory actions of manidipine and its modulation of the systemic and renal effects of endothelin We examined the actions of intravenously administered manidipine on systemic and renal microcirculatory hemodynamics and its efficacy in antagonizing endothelin-1 (ET-1)-evoked by: 2 The Respiratory System N.
Edward Robinson KEY POINTS 1. The horse’s unique anatomy, size, and weight make it particularly vulnerable to anesthetic. Atenolol is a cardioselective beta-blocker that is widely used in the treatment of hypertension and angina pectoris. Atenolol has been linked to rare cases of drug induced liver injury, some of which have been fatal.
Atenolol is an ethanolamine compound having a (4-carbamoylmethylphenoxy)methyl group at the 1-position and an N-isopropyl. Full text of "Journal of the Mississippi State Medical Association" See other formats.
Journal of Special Operations Medicine - Article Index. The Journal of Special Operations medicine peer-reviewed article index displays all of our articles listed in alphabetical order. Simply click on the article that interests you, and you will be taken to the abstract for that article.
This banner text can have markup. web; books; video; audio; software; images; Toggle navigation. FAA/AM/11 - Effect of glyceryl trinitrate on pulmonary vasculature of anesthetized dogs. FAA/AM/12 - Comments on progress in ballistocardiographic research and the current state of the art.
FAA/AM/13 - The size cue to visually perceived distance. FAA/AM/14 - Effects of bilateral caloric habituation on nystagmus responses of the cat. Methods: Female Yorkshire swine (n =15) were anesthetized and cannulated with a 23Fr dual-lumen catheter.
Venovenous extracorporeal life support (VV ECLS) was initiated via the right jugular vein and carried out with animals uninjured on day 1 and injured by bilateral pulmonary contusion on day 2. Professor Angus has been an Honorary Professorial Fellow and Professor Emeritus of the Department of Pharmacology and Therapeutics, Faculty of Medicine, Dentistry and Health Sciences at the University of Melbourne since From toProf.
Angus was Dean of the Faculty of Medicine, Dentistry and Health Angus earned a Bachelor of Science in pharmacology with Honours. InWilliam Murrell, a physician and practical therapist in London, introduced glyceryl trinitrate as a remedy for angina pectoris . Since then NO donors have been used to treat a variety of disorders including angina pectoris, coronary artery disease, congestive heart failure and hypertension.
Glyceryl trinitrate in therapeutic doses may produce: C A. Dilation of cerebral blood vessels B. Reflex tachycardia C. Both A & B D. Neither A nor B A chronic hypertensive man, during his recent visit to the hospital A had his antihypertensive medication changed from Tenormin to Zestril.
He develops a dry cough after a period of 2 weeks. His.Mean pulmonary artery pressure (MAP) increased about 6 mmHg in Controls, about 4 mmHg in NMAcz low and high and about 2 mmHg in Acz. The pulmonary vascular resistance increased by dynscm-5 in Controls, by dyn s cm-5 in NMAcz (low), by dynscm-5 in NMAcz (high), and by dynscm-5 in Acz.Furthermore, the chapter also provides an overview on the cellular and molecular mechanisms involved in PPARgamma-mediated inhibitory effect on pulmonary vascular remodeling, a major contributor to the elevated pulmonary vascular resistance in patients with pulmonary arterial hypertension.
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